Coffee A Potent and Functional Food in Reduced Risk and Treatment of Hyperglycemia

Hyperglycemia is a condition characterized by abnormally high blood glucose in the bloodstream.

Coffee, a popular and social beverage all over the world, particularly in the West, is a drink made from roasted bean from the Coffea plant, native to tropical Africa and Madagascar.

According to the Nagoya University, the study of antidiabetic effect of coffee and caffeine in spontaneously diabetic KK-A(y) mice, given regular drinking water (controls) or 2-fold diluted coffee for 5 weeks, suggested that group treatment with coffee and coffee caffeine expressed an ameliorated development of hyperglycemia and improved insulin sensitivity in compared to water treatment group.

The efficacy of coffee and coffee caffeine in reduced risk of hyperglycemia may also be attributed to the regulation of white adipose tissue mRNA levels in elevated the production of anti-inflammatory cytokines.

Furthermore, researchers in the differentiation of the liver activity also found that although similar in treatment
1. Coffee injection improved the fatty liver and
2. Coffee caffeine exhibited the ameliorated hyperglycemia and improved fatty liver.

In further to strengthen the result of the above analysis, the Nagoya University and other institution conducted a study related to the effect of coffee on pancreatic β-cells against induction of diabetes by streptozotocin (STZ), indicated that mice treated with coffee expressed a lowered or significantly lowered serum glucose concentration is compared to water ingestion group.

Dr. Kobayashi M, the lead researcher said, "coffee ingestion also suppressed the deterioration of hyperglycemia in diabetic mice which had been already injected with STZ" and "coffee ingestion prevented the development of STZ-induced diabetes and suppressed hyperglycemia in STZ-diabetic mice. Caffeine or decaffeinated coffee ingestion did not significantly suppress STZ-induced hyperglycemia".

Other, in the study of postprandial hyperglycemia as a direct and independent risk factor for cardiovascular diseases,s showed that injection of coffee polyphenol may improve postprandial hyperglycemia and vascular endothelial function by increasing GLP-1 release in affecting gut motility, inhibit gastric acid secretion, and inhibit glucagon secretion. and/or reducing oxidative stress.

More importantly, in a total of Nineteen subjects consumed a test meal with either placebo- or extract coffee polyphenol CPE-containing beverage, researchers showed that treatment group displayed a significantly lower peak postprandial increase in blood glucose in compared to placebo beverage group.

According to the correlation analysis, the treatment group also improved impaired Fibromuscular dysplasia (FMD) postprandial response in associated with blood glucose increase after injection of the CPE beverage.

Taking together, there is no doubt that coffee may be considered as a functional food used as therapeutic substances in reduced risk and treatment of hyperglycemia.


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Author Biography
Kyle J. Norton (Scholar, Master of Nutrition, All right reserved)
Health article writer and researcher; Over 10.000 articles and research papers have been written and published online, including worldwide health, ezine articles, article base, health blogs, self-growth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bioscience, ISSN 0975-6299.

Sources
(1) Coffee and caffeine ameliorate hyperglycemia, fatty liver, and inflammatory adipocytokine expression in spontaneously diabetic KK-Ay mice by Yamauchi R1, Kobayashi M, Matsuda Y, Ojika M, Shigeoka S, Yamamoto Y, Tou Y, Inoue T, Katagiri T, Murai A, Horio F.(PubMed)
(2) Coffee Ingestion Suppresses Hyperglycemia in Streptozotocin-Induced Diabetic Mice by Kobayashi M1, Kurata T1, Hamana Y1, Hiramitsu M2, Inoue T2, Murai A1, Horio F1.(PubMed)