1/2 cup banana
1/4 cup yogurt
1/4 plain chocolate
1 cup vitamin D fortified low fat milk
The finding of a natural source for treatment of Gitelman syndrome has been running into many obstacles, many ingredients showed initially with promising result in animal studies have not produced same potentials in large sample size and mutli centers human trials.
Gitelman syndrome is an autosomal recessive distal renal tubular disorder caused by defective sodium chloride transporters inducing hypokalaemic metabolic alkalosis, hypomagnesaemia and hypocalciuria(1). In other words it is a kidney disease caused by an imbalance of ions of potassium, magnesium, and calcium. Recent study lead by the University of Padova suggested that patients with Gitelman syndrome are associated to increase existence of reactive oxygen species (ROS) and oxidative stress of that status could offer chances for new therapeutic targets in disease characterized by high levels of reactive oxygen species(2).
According to the Royal Liverpool and Broadgreen University Hospitals NHS Trust. treatment of Gitelman syndrome may include oral potassium supplement, weekly intravenous potassium and magnesium infusion, correction of vitamin D level and the offering of appropriate dietary advice(3).
The combination of Avocado, Banana, Yogurt, Plain Chocolate and Vitamin D fortified low fat milk smoothie may hold a key in further studies in production of an effective natural ingredients for treatment of Gitelman syndrome without inducing adverse effects.
People with Gitelman syndrome should drink as much as they can depending to the digestive toleration.
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(1) Management of a severe case of Gitelman syndrome with poor response to standard treatment b Koudsi L1, Nikolova S1, Mishra V1.(PubMed)
(2) Bartter/Gitelman syndromes as a model to study systemic oxidative stress in humans by Maiolino G1, Azzolini M1, Rossi GP1, Davis PA2, Calò LA3(PubMed)
(3) Management of a severe case of Gitelman syndrome with poor response to standard treatment by Koudsi L1, Nikolova S1, Mishra V1.(PubMed)